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As we age or deal with chronic stress, the natural production of these signals drops

These compositional disruptions contribute to increased intestinal permeability and sustained peripheral inflammation, creating conditions conducive to -synuclein misfolding within the enteric nervous system and its subsequent propagation along gutbrain pathways [25]

Cholinergic neurons of the enteric nervous system, which interact closely with gastrointestinal epithelial cells, as well as ACh signaling in non-neuronal cells such as enteric glia and pancreatic cells, depend on the essential role of thiamine in ACh production and the facilitation of ACh signaling

Microbial composition predicts genital tract inflammation and persistent bacterial vaginosis in South African adolescent females