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In both AKI and CKD models, mitochondrial biogenesis is greatly impaired, leading to the insufficient supply of ATP and cell injury in various cell types, particularly in TECs in kidney, (Galvan et al., 2017

This is one of the sneakiest parts of GLP-1 fatigue

What this evidence does show: in rat models of acute, surgically induced tendon and ligament injury, BPC-157 administration is associated with improved healing endpoints versus saline controls across the labs that have studied it