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The molecular tweezer CLR01 inhibits -syn aggregation through binding to lysine residues of -syn that are crucial for its oligomerization, and it has shown efficacy, in terms of motor symptom improvement and decreased oligomeric -syn burden, in transgenic mouse models of PD (Bengoa-Vergniory et al

developed a drug-delivery system loaded with temozolomide based on porous silicon NPs to improve the survival of mice with glioma

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