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In parallel, metabolic interventions, including high-calorie diets and medium-chain triglyceride supplementation, are undergoing clinical evaluation to counteract the hypercatabolic state characteristic of ALS [79], a systemic reflection of the severe energetic crisis driven by motor neuron mitochondrial failure

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Glycophorin-A is visible on the morphologically identifiable erythroid precursors subsequently the colony-forming unit erythroid (CFU-E) stage, and reaches its greatest expression at the late normoblast stage

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