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It is a natural feedback loop that has kept humans from overeating for millennia

doi:10.3389/fnut.2021.614105

If the metabolism of ethanol in other organs is not reduced by GLP-1R agonists, then it is possible that the elevated blood levels caused by reduced hepatic metabolism could have detrimental effects on other organs, shifting ethanol metabolism and production of acetaldehyde from the liver to other organs

Figure 3: Acetaldehyde, a byproduct of alcohol, disrupts the balance of SAM and SAH, increasing homocysteine (Hcy) conversion to methionine while reducing cysteine and glutathione (GSH) production