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Indeed, the combination of D + Q is more effective and allows to target more senescent cell types (compared to the use of each drug alone), by inhibiting the pro-survival pathways induced in senescent cells, notably in idiopathic pulmonary fibrosis (IPF) 10,11,12 , diabetes and chronic kidney disease 13 or Alzheimers disease 14

Further information from the literature, CRISPR-Cas studies 32,33,34 , and the availability of suitable inhibitors were additionally used to identify candidate targets suitable for experimental testing

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