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& Kimmel, A
Thus, the effect of exogenous T4 treatment on inhibition of microglial activation after damage may contribute to reduce the secretion of inflammatory mediators (Zhang et al., 2016b, Zhou et al., 2015), and thereby prevent and/or reduce damage of OLs after EAE by attenuating immune onslaught (neuroprotection)
These studies have generally demonstrated positive therapeutic outcomes, but translation to clinical applications requires careful consideration of species differences and experimental limitations
The recommended dosage of praziquantel varies according to body weight
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