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Mechanisms include: Restoration of endothelial nitric oxide synthase (eNOS) Upregulation of VEGF and FGF-2 (fibroblast growth factor 2) Reduction of oxidative stress and cytokine-induced apoptosis BPC-157 preserved endothelium-dependent vasodilation via upregulation of eNOS and suppression of vascular inflammation. Gwyer Findlay et al., Peptides 4

Note, the regulation of the brain blood flow was ascribed to BPC 157 therapy in occlusion/occlusion-like syndrome (including also in amphetamine rats) given rapid decrease in the brain swelling and counteracted intracranial hypertension along with activation of the collateral rescuing pathways immediately upon BPC 157 therapy [40,41,42,43,47,48,49,50,51,52,53,54,55,56]

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