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In response to pathological stress such as sepsis, microglia undergo rapid activation, releasing inflammatory factors and generating oxygen free radicals, which contribute to neuronal dysfunction and synaptic impairment [11]

After 4 days of activation with anti-IgM, CD40 ligand, and IL-4, we found that blocking of mitochondrial metabolism with mitochondrial inhibitors or blocking GSH synthesis by BSO had lowered antibody production of FoB in a dose-dependent manner (Fig

Chelation involves administering EDTA, a compound that binds to heavy metals in the body, allowing them to be excreted through the kidneys

Cancer 29 , 831839 (1993)