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This was corroborated by observations of embryonic lethality in mouse and Caenorhabditis elegans models, with oxidative stress damage shown to be the underlying cause (31)

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2 Takaoka et al stated that the expression of estrogen receptor (ER) in ovarian endometrial stromal cells (OESCs) was suppressed at the gene and protein levels after DRIAs treatment, which in turn inhibited the expression of COX-2 and the production of PGE2

This abnormal visceral sensory pathway is closely related to central sensitization within the nucleus tractus solitarius-thalamus-cerebral cortex pathway