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The anti-inflammatory effects of methylsulfonylmethane on lipopolysaccharide-induced inflammatory responses in murine macrophages, 14
(2014, Nature Medicine) established NNMT as a major regulator of metabolic rate in white adipose tissue, showing that NNMT knockdown in mice produced a lean phenotype with elevated energy expenditure providing the foundational rationale for NNMT inhibition as a metabolic research target
Concerning the AR content in skeletal muscle cells, very conflicting results have been previously obtained by AR mRNA analysis in young and adult males, under basal conditions or after exercise [70,71,72,73,74]
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