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GLP-1 treatment protects against EndMT through inhibiting the activation of poly (ADP-ribose) polymerase 1 (PARP-1) which can elicit Smad3 and transforming growth factor (TGF)-1 responses

Research on AD models has suggested that reactive astrocytes are more susceptible to neuronal reprogramming than their nonreactive counterparts, underscoring the role of astrocytic heterogeneity in influencing cell differentiation 25,149

doi: 10.1016/S0006-8993(00)02816-X